By Jennifer I. Lim (ed.)
Addressing a big situation affecting hundreds of thousands all over the world, this resource compiles the main functional and groundbreaking examine at the etiology, evaluation, and remedy of Age-Related Macular Degeneration (AMD). With full-color illustrations all through, this reference explores examine developments that experience ended in novel cures that supply sight saving, much less damaging different types of therapy for exudative AMD, in addition to suggestions to avoid the development of non-exudative AMD. With 3 new chapters and expansive volume of part updates, this resource offers the newest experiences on OCT imaging, experimental remedies, new functions for thermal lasers, and gene treatment for AMD. The resource additionally summarizes stories from present medical trials to stay the surest within the provision of deal with sufferers with this disabling disorder.
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Addressing an immense situation affecting hundreds of thousands around the world, this resource compiles the main sensible and groundbreaking learn at the etiology, review, and therapy of Age-Related Macular Degeneration (AMD). With full-color illustrations all through, this reference explores learn developments that experience ended in novel remedies that provide sight saving, much less harmful types of remedy for exudative AMD, in addition to ideas to avoid the development of non-exudative AMD.
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Additional resources for Age-Related Macular Degeneration
Statistical evidence has been generated to suggest that infection with various infectious agents, especially Chlamydia pneumoniae or cytomegalovirus (CMV), might initiate vascular injury and explain the systemic inﬂammatory signs (122–125). Numerous epidemiologic studies have revealed a statistical correlation between atherosclerosis and serologic evidence of infection with C. pneumoniae (122). Follow-up studies have demonstrated the presence of C. pneumoniae by histochemical methods within atherosclerotic plaques and organisms have been cultured from the lesions (125).
Enzymatic cleavage) remain unknown. Finally, oxidants have been implicated as important mediators and ampliﬁers in progression of renal disease (173). Direct Evidence for Innate or Antigen-Specific Immune Effectors Direct evidence for the role of immune mechanism in AMD is scant. The best data suggest an important role for macrophage-mediated innate immunity (22,50–58). Investigators have observed that choroidal macrophages appear to be important in the pathogenesis of both early and late AMD. However, macrophage involvement is clearly different than their participation in overt inﬂammatory disorders characterized by widespread cellular inﬁltration.
Many forms of chronic glomerulonephritis are caused by antibody-dependent mechanisms, and some of these disorders are characterized by subendothelial or subepithelial deposit formation (102,162–164). Direct deposition of circulating antibodies targeted at antigens uniformly expressed within the glomerular matrix is a welldeﬁned but rare form of glomerulonephritis, especially in Goodpasture’s syndrome. Deposition of preformed circulating antigen/antibody complexes in the blood has been proposed as another major mechanism in many types of glomerulonephritis associated with deposit formation.
Age-Related Macular Degeneration by Jennifer I. Lim (ed.)