By James C. Bobrow, MD
This part underwent significant revision for the 2008-2009 variation. part eleven reports the anatomy, body structure, embryology, and pathology of the lens. It additionally covers the epidemiology of cataracts and their overview and administration in adults. additionally, an summary of lens and cataract surgical procedure is supplied, issues of cataract surgical procedure are mentioned, and cataract surgical procedure in precise occasions is explored. includes many new photographs.
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Additional resources for 2008-2009 Basic and Clinical Science Course: Section 11: Lens and Cataract (Basic and Clinical Science Course 2008-2009)
Duane's Clinical Ophthalmology. 1-20. Streeten BW. Zonular apparatus; Saunders; 2000: 1355-1408. Vol!. Philadelphia: Lippincott; 2002:chap 71A, pp Worgul BY. The lens. In: Jakobiec FA, ed. Oct/lar Anatomy, Embryology. and Teratology. Philadelphia: Harper & Row; 1982:331-353. Congenital Anomalies and Abnormalities Congenital Aphakia The lens is absent in congenital aphakia, a very rare anomaly. Two forms of congenital aphakia have been described. In primary aphakia, the lens placode fails to form from the surface ectoderm in the developing embryo.
CHAPTER 5 Pathology Aging Changes As the lens ages, it increases in weight and thickness and decreases in accommodative power. As new layers of cortical fibers are formed concentrically, the lens nucleus undergoes compression and hardening (nuclear sclerosis). Chemical modification and proteolytic cleavage of crystallins (lens proteins) result in the formation of high-molecular-weight protein aggregates. These aggregates may become large enough to cause abrupt fluctuations in the local refractive index of the lens, thereby scattering light and reducing transparency.
Marfan Syndrome Marfan syndrome is a heritable disorder with ocular, cardiac, and skeletal manifestations. Though usually inherited as an autosomal dominant trait, the disorder appears with no family history in approximately 15% of cases. Marfan syndrome is believed to result from an abnormality of fibrillin, a connective tissue component. Affected individuals are tall, with arachnodactyly (Fig 4-15A) and chest wall deformities. Associated cardiac abnormalities include dilated aortic root and mitral valve prolapse.
2008-2009 Basic and Clinical Science Course: Section 11: Lens and Cataract (Basic and Clinical Science Course 2008-2009) by James C. Bobrow, MD